截止目前，引用Bioss產(chǎn)品發(fā)表的文獻(xiàn)共26093篇，總影響因子123723.87分，發(fā)表在Nature, Science, Cell以及Immunity等頂級期刊的文獻(xiàn)共60篇，合作單位覆蓋了清華、北大、復(fù)旦、華盛頓大學(xué)、麻省理工學(xué)院、東京大學(xué)以及紐約大學(xué)等國際知名研究機(jī)構(gòu)上百所。
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近期收錄2023年8月引用Bioss產(chǎn)品發(fā)表的文獻(xiàn)共304篇（圖一，綠色柱），文章影響因子(IF) 總和高達(dá)1925.1，其中，10分以上文獻(xiàn)36篇（圖二）。
圖一
圖二
本文主要分享引用Bioss產(chǎn)品發(fā)表文章至Nature Nanotechnology, Immunity, Cancer Cell等期刊的4篇 IF＞15 的文獻(xiàn)摘要，讓我們一起欣賞吧。
Nature [IF=64.8]
文獻(xiàn)引用產(chǎn)品：bs-0737R-AF488
HIF-1 Alpha/AF488 pAb | FCM
作者單位：哈佛大學(xué)醫(yī)學(xué)院
文獻(xiàn)引用抗體：bs-0086R
TGF beta 1 Rabbit pAb | IF
作者單位：賓夕法尼亞大學(xué)
摘要：The desmoplastic stroma in solid tumors presents a formidable challenge to immunotherapies that rely on endogenous or adoptively transferred T cells, however, the mechanisms are poorly understood. To define mechanisms involved, here we treat established desmoplastic pancreatic tumors with CAR T cells directed to fibroblast activation protein (FAP), an enzyme highly overexpressed on a subset of cancer-associated fibroblasts (CAFs). Depletion of FAP+ CAFs results in loss of the structural integrity of desmoplastic matrix. This renders these highly treatment-resistant cancers susceptible to subsequent treatment with a tumor antigen (mesothelin)-targeted CAR T cells and to anti-PD-1 antibody therapy. Mechanisms include overcoming stroma-dependent restriction of T cell extravasation and/or perivascular invasion, reversing immune exclusion, relieving T cell suppression, and altering the immune landscape by reducing myeloid cell accumulation and increasing endogenous CD8+ T cell and NK cell infiltration. These data provide strong rationale for combining tumor stroma- and malignant cell-targeted therapies to be tested in clinical trials.
Nature Communications [IF=16.6]
文獻(xiàn)引用抗體：D10746
Fibrinogen (from Rat plasma)
作者單位：四川大學(xué)
BRAIN BEHAVIOR AND IMMUNITY [IF=15.1]
文獻(xiàn)引用產(chǎn)品：bs-0480R
IFN gamma Rabbit pAb | IF
作者單位：康涅狄格大學(xué)
摘要：
Background
Alzheimer’s disease (AD) is the most common cause of dementia in older adults and characterized by progressive loss of memory and cognitive functions that are associated with amyloid-beta (Aβ) plaques and neurofibrillary tangles. Immune cells play an important role in the clearance of Aβ deposits and neurofibrillary tangles. T cells are the major component of the immune system. The thymus is the primary organ for T cell generation. T cell development in the thymus depends on thymic epithelial cells (TECs). However, TECs undergo both qualitative and quantitative loss over time. We have previously reported that a recombinant (r) protein containing FOXN1 and a protein transduction domain can increase the number of TECs and subsequently increases the number of T cells in mice. In this study we determined the ability of rFOXN1 to affect cognitive performance and AD pathology in mice.
Methods
Aged 3xTg-AD and APP/PS1 AD mice were injected with rFOXN1 or control protein. Cognitive performance, AD pathology, the thymic microenvironment and immune cells were then analyzed.
Results
Administration of rFOXN1 into AD mice improves cognitive performance and reduces Aβ plaque load and phosphorylated tau in the brain...
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